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Causes of retinopathy include but are not limited to:
• Diabetes mellitus, which causes diabetic retinopathy
• Arterial hypertension, which causes hypertensive retinopathy
• Retinopathy of prematurity due to prematurity of the newborn (under the 9 months of human pregnancy).
• Radiation retinopathy due to exposure to ionizing radiation.
• Solar retinopathy due to direct sunlight exposure.
• Sickle cell disease
• Retinal vascular disease such as retinal vein or artery occlusion.
• Trauma, especially to the head, and several diseases may cause Purtscher's retinopathy
• Hyperviscosity-related retinopathy as seen in disorders which cause paraproteinemia
Many types of retinopathy are proliferative, most often resulting from neovascularization or blood vessel overgrowth. Angiogenesis is the hallmark precursor that may result in blindness or severe vision loss, particularly if the macula becomes affected.
Retinopathy may more rarely be due to ciliopathic genetic disorders, such as Alström syndrome or Bardet–Biedl syndrome.
Retinopathy is diagnosed by an ophthalmologist or an Optometrist during eye examination. Treatment depends on the cause of the disease.

Treatment is based on the cause of the retinopathy and may include laser therapy to the retina. In recent years targeting the pathway controlling vessel growth or angiogenesis has been promising. Vascular endothelial growth factor (VEGF) seems to play a vital role in promoting neovascularization. Using anti-VEGF drugs (antibodies to sequester the growth factor), researches have shown significant reduction in the extent of vessel outgrowth. Several anti-VEGF treatments are currently under review in both clinical trials and in preclinical labs.